[PDF][PDF] Making new connections: role of ERK/MAP kinase signaling in neuronal plasticity
S Impey, K Obrietan, DR Storm - Neuron, 1999 - cell.com
S Impey, K Obrietan, DR Storm
Neuron, 1999•cell.comOne of the most intriguing questions in neurobiology GAP, Ca2+/DAG GTP exhange factors
[GEFs], NF1, is how neurons and synapses encode the long-term N-Ras, and B-Raf) is
largely restricted to the CNS. An changes in synaptic efficacy that underlie memory coninitial
clue to the role of the ERK/MAPK cascade in the solidation. A recent set of studies suggests
that the CNS came from the observation that expression of ERK/mitogen-activated protein
kinase (MAPK) plays a
[GEFs], NF1, is how neurons and synapses encode the long-term N-Ras, and B-Raf) is
largely restricted to the CNS. An changes in synaptic efficacy that underlie memory coninitial
clue to the role of the ERK/MAPK cascade in the solidation. A recent set of studies suggests
that the CNS came from the observation that expression of ERK/mitogen-activated protein
kinase (MAPK) plays a
One of the most intriguing questions in neurobiology GAP, Ca2+/DAG GTP exhange factors [GEFs], NF1, is how neurons and synapses encode the long-term N-Ras, and B-Raf) is largely restricted to the CNS. An changes in synaptic efficacy that underlie memory coninitial clue to the role of the ERK/MAPK cascade in the solidation. A recent set of studies suggests that the CNS came from the observation that expression of ERK/mitogen-activated protein kinase (MAPK) plays a
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